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Athletic heart syndrome, also knows as athlete's heart, is a medical syndrome in which the human heart is enlarged due to excessive amounts of exercise. Common in athletes who exercise more than an hour almost every day and occasionally heavy weight trainers, this syndrome is believed to be a benign conditional but may be hard to distinguish from other serious medical conditions.
Description[edit]
During intensive, prolonged endurance and strength training, the body signals the heart to pump more blood through the body to counteract the oxygen deficit building in the skeletal muscles. Enlargement of the heart is a natural physical adaptation of the body to deal with the high pressures and large amounts of blood that can affect the heart during these periods of time. After time, these pressures cause the muscle mass, wall thickness, and chamber size of the left ventricle of the heart to increase.[1]
Cardiac output, the amount of blood that leaves the heart, is proportional to both the chamber sizes of the heart and the rate at which the heart beats. With a larger left ventricle, the heart rate can decrease and still maintain a level of cardiac output necessary for the body. Therefore, it is very common for athletes to have lower resting heart rates than nonathletes.
Symptoms and Diagnosis[edit]
There are no symptoms for people with athletic heart syndrome, although an indicator would be a consistently low resting heart rate. AHS is usually diagnosed during a routine screening or during tests for other medical issues. An enlarged heart can be seen on an echocardiogram or sometimes a chest X-ray. Dur to the similarities between AHS and more serious cardiac problems, an electrocardiogram (EKG) and exercise stress tests are sometimes performed.
The EKG can detect sinus bradycardia, a resting heart rate of fewer than 60 beats per minute. This is often accompanied by sinus arrhythmia. The heartbeat of a person with AHS can sometimes be irregular while at rest, but usually returns to normal after exercise begins.
One common cardiovascular disease that has similar EKG reads is hypertrophic cardiomyopathy, which is characterized by the thickening of the heart's walls. This genetic disorder is found in 1 out of 500 Americans and is responsible for thousands of sudden death cases every year. Of all sudden death cases, only about 8% are exercise related. [2] The following table shows distinguishing characteristics of the two conditions.
| Feature | Athletic Heart Syndrome | ! Cardiomyopathy |
|---|---|---|
| Left ventricular hypertrophy | < 13 mm | > 15 mm |
| Left ventricular end-diastolic diameter | < 60 mm | > 70 mm |
| Diastolic function | Normal (E:A ratio > 1) | Abnormal (E:A ratio < 1) |
| Septal hypertrophy | Symmetric | Asymmetric (in hypertrophic cardiomyopathy) |
| Family history | None | May be present |
| BP response to exercise | Normal | Normal or reduced systolic BP response |
| Deconditioning | Left ventricular hypertrophy regression | No left ventricular hypertrophy regression |
Features Distinguishing Athletic Heart Syndrome From Cardiomyopathy [3]
Prognosis and Treatment[edit]
Although the heart experiences structural changes (i.e. wall thickening) that are common with some cardiac disease, no unfavorable effects are evident. 80% of people affected by this syndrome show a decrease in such structural changes and bradycardia with detraining. Unfortunately, the lack of long-term data limits our ability to determine whether or not the remaining 20% of people who do indeed have residual chamber enlargement have experienced any negative side effects.
No treatment is required for people with athletic heart syndrome. In some cases, 3 months of deconditioning are required in order ti distinguish AHS from cardiomyopathy, though this may be met with resistance since training is an integral part of an athlete's life.
See also[edit]
References[edit]
- ^ Lohr, John Thoman. (1999). Athletic Heart Syndrome. Gale Encyclopedia of Medicine.
- ^ Alexander, R. W., R. C. Schlant, and V. Fuster, editors. The Heart. 9th ed. New York: McGraw-Hill, 1998.
- ^ Merck Manual Professional. (2005). Athletic Heart Syndrome. Retrieved March 16, 2008 from http://www.merck.com/mmpe/sec07/ch082/ch082c.html.