File:INO80 stabilizes replication forks and counteracts mislocalization of H2A.Z.png
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Summary
| Description |
English: Excess H2A.Z can cause replication defects and genome instability in the absence of the INO80 chromatin remodeler, which facilitates the removal of H2A.Z from chromatin. Aberrant H2A.Z accumulation may alter the epigenetic landscape at sites of replication stress.[1] |
| Date | |
| Source | https://doi.org/10.3390/genes6030858 |
| Author | Simran Khurana, and Philipp Oberdoerffer |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 23:43, 24 May 2020 | | 2,550 × 1,600 (709 KB) | Rob Hurt | Uploaded a work by Simran Khurana, and Philipp Oberdoerffer from https://doi.org/10.3390/genes6030858 with UploadWizard |
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